首页> 外文OA文献 >The Predominant Pathway of Apoptosis in THP-1 Macrophage-Derived Foam Cells Induced by 5-Aminolevulinic Acid-Mediated Sonodynamic Therapy is the Mitochondria-Caspase Pathway Despite the Participation of Endoplasmic Reticulum Stress

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The Predominant Pathway of Apoptosis in THP-1 Macrophage-Derived Foam Cells Induced by 5-Aminolevulinic Acid-Mediated Sonodynamic Therapy is the Mitochondria-Caspase Pathway Despite the Participation of Endoplasmic Reticulum Stress

机译：5-氨基乙酰丙酸介导的动力学疗法诱导THp-1巨噬细胞源性泡沫细胞凋亡的主要途径是线粒体 - 半胱氨酸蛋白酶途径尽管参与了内质网应激

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Huan Wang; Yang Yang; Haibo Chen; Juhua Dan; Jiali Cheng; Shuyuan Guo; Xin Sun; Wei Wang; Yiwei Ai; Shuchang Li; Zhixin Li; Li Peng; Zhen Tian; Liming Yang; Jichao Wu; Xin Zhong; Qi Zhou; Peng Wang; Zhiguo Zhang; Wenwu Cao; Ye Tian;
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1. The Predominant Pathway of Apoptosis in THP-1 Macrophage-Derived Foam Cells Induced by 5-Aminolevulinic Acid-Mediated Sonodynamic Therapy is the Mitochondria-Caspase Pathway Despite the Participation of Endoplasmic Reticulum Stress [J] . Ai Y., Cao W., Chen H., Cellular Physiology and Biochemistry . 2014,第6期

机译：尽管参与了内质网应激，但5-氨基乙酰丙酸介导的声波动力学疗法诱导的THP-1巨噬细胞衍生泡沫细胞凋亡的主要途径是线粒体-半胱天冬酶途径。
2. miR-7112-3p targets PERK to regulate the endoplasmic reticulum stress pathway and apoptosis induced by photodynamic therapy in colorectal cancer CX-1 cells [J] . Photodiagnosis and Photodynamic Therapy . 2020,第Mara期

机译：miR-7112-3p靶向PERK调节大肠癌CX-1细胞中光动力疗法诱导的内质网应激途径和细胞凋亡
3. Endoplasmic reticulum stress could induce autophagy and apoptosis and enhance chemotherapy sensitivity in human esophageal cancer EC9706 cells by mediating PI3K/Akt/mTOR signaling pathway [J] . Fang Zhou, Yan-Hua Li, Jian-Jun Wang, Tumour biology : . 2017,第6期

机译：内质网应激可通过介导PI3K / Akt / mTOR信号通路诱导食管癌EC9706细胞自噬和凋亡并增强化疗敏感性
4. Endoplasmic Reticulum Stress Signaling Pathways Is Involved in Trichosanthin-Induced Apoptosis in Human Cervical Cancer Cell Line Hela [C] . Huang Yiling, Huang Liming, You Chengcheng, 2010 4th International Conference on Bioinformatics and Biomedical Engineering . 2010

机译：内质网应激信号通路涉及天花粉蛋白诱导的人宫颈癌细胞株Hela的凋亡。
5. Modulation of NF-kappaB and induction of endoplasmic reticulum stress potentiate chemotherapy-induced apoptosis in oral squamous cell carcinoma. [D] . Fribley, Andrew M. 2005

机译：口腔鳞状细胞癌中NF-κB的调节和内质网应激的诱导增强了化疗诱导的细胞凋亡。
6. 5-Aminolevulinic Acid-Mediated Sonodynamic Therapy Inhibits RIPK1/RIPK3-Dependent Necroptosis in THP-1-Derived Foam Cells [O] . Fang Tian, Jianting Yao, Meng Yan, -1

机译：5-氨基乙酰丙酸介导的声波动力学疗法抑制THP-1衍生的泡沫细胞中RIPK1 / RIPK3依赖性坏死病。
7. GW25-e0820 Apoptosis of THP-1 macrophage-derived foam cells induced by 5-aminolevulinic acid-mediated sonodynamic therapy is mitochondria-caspase pathway predominant despite the participation of endoplasmic reticulum stress [O] . Huan Wang, Tian Ye 2014

机译：GW25-e0820尽管内质网应激参与，但5-氨基乙酰丙酸介导的声波动力学疗法诱导的THP-1巨噬细胞源性泡沫细胞的凋亡仍是主要的线粒体-半胱天冬酶途径。

The Predominant Pathway of Apoptosis in THP-1 Macrophage-Derived Foam Cells Induced by 5-Aminolevulinic Acid-Mediated Sonodynamic Therapy is the Mitochondria-Caspase Pathway Despite the Participation of Endoplasmic Reticulum Stress

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